Eat a diet high in oil and processed foods Processed foods, especially those high in sugar, salt, refined carbohydrates, and saturated fat, are associated with an increased risk of developing NAFLD (no alcohol fatty liver disease).
A diet rich in these ingredients can lead to weight gain, insulin resistance, and fat accumulation in the liver.Research published in the journal Clinical Nutrition in 2018 shows that excessive consumption of these foods can contribute to the development of NAFLD.
Reduce the consumption of processed foods.Limit foods high in sugar, salt, and saturated fat.Increase fiber-rich foods, eat plenty of green vegetables, fruits, and whole grains to support liver function.Follow a balanced diet and exercise regularly to maintain a healthy weight.
Drink alcohol regularly According to the International Agency for Research on Cancer (IARC), alcohol is classified as a type 1 carcinogen, meaning it has clear evidence of the ability to cause cancer in humans.
Alcohol consumption is directly linked to cancers such as oral cancer, phlegm, and lai vay (due to alcohol damaging the lining of the mouth and throat, creating conditions for cancer agents to invade and develop), esophageal cancer (due to damage to the lining and the formation of carcinogens during alcohol metabolism), liver cancer (due to liver inflammation, cirrhosis).
Alcohol consumption also increases the risk of breast cancer in women.
Increased insulin resistance Increased insulin resistance is an important factor in the development of many metabolic diseases, especially type 2 diabetes.
Insulin resistance occurs when cells in the body do not respond effectively to insulin, leading to the body needing to produce more insulin to maintain normal blood sugar levels.
Insulin helps increase the absorption of glucose into muscle cells for use as energy. When insulin resistance occurs, this ability is reduced, leading to a buildup of glucose in the blood. In addition, insulin inhibits fat breakdown. When there is insulin resistance, this process is interrupted, leading to increased release of free fatty acids into the blood, contributing to dyslipidemia and increasing the risk of fatty liver.
